Acute hemiconvulsive encephalopathy of childhood with prominent unilateral delta activity in the electroencephalogram

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Abstract

Twenty children with an acute hemiconvulsive encephalopathy are reported who had a sudden onset of focal epileptic seizures. The majority had clinical evidence of hemiparesis which was transient. All had very prominent unilateral delta activity in the EEG which also cleared gradually but at a rate much slower than the clinical symptoms. The initial picture of these children was similar to those with post-convulsive acute hemiplegia of childhood but the subsequent course was benign with regards to neurological deficit, intellectual performance and recurrence of seizures. It is emphasized that prompt control of convulsions in such patients may prevent further cerebral damage secondary to the seizures themselves which may have been responsible for poor prognosis previously reported in similar patients.

Original languageEnglish
Pages (from-to)137-148
Number of pages12
JournalThe Indian Journal of Pediatrics
Volume48
Issue number2
DOIs
StatePublished - Mar 1981

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Brain Diseases
Electroencephalography
Seizures
Hemiplegia
Paresis
Epilepsy
Recurrence

Keywords

  • Acute hemiconvulsive encephalopathy
  • Acute hemiplegia of childhood
  • H.H.E. syndrome

ASJC Scopus subject areas

  • Pediatrics, Perinatology, and Child Health

Cite this

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AB - Twenty children with an acute hemiconvulsive encephalopathy are reported who had a sudden onset of focal epileptic seizures. The majority had clinical evidence of hemiparesis which was transient. All had very prominent unilateral delta activity in the EEG which also cleared gradually but at a rate much slower than the clinical symptoms. The initial picture of these children was similar to those with post-convulsive acute hemiplegia of childhood but the subsequent course was benign with regards to neurological deficit, intellectual performance and recurrence of seizures. It is emphasized that prompt control of convulsions in such patients may prevent further cerebral damage secondary to the seizures themselves which may have been responsible for poor prognosis previously reported in similar patients.

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