APOL1,α-thalassemia, and BCL11A variants as a genetic risk profile for progression of chronic kidney disease in sickle cell anemia

Santosh L. Saraf, Binal N. Shah, Xu Zhang, Jin Han, Bamidele O. Tayo, Taimur Abbasi, Adam Ostrower, Elizabeth Guzman, Robert E. Molokie, Michel Gowhari, Johara Hassan, Shivi Jain, Richard S. Cooper, Roberto F. Machado, James P. Lash, Victor R. Gordeuk

Research output: Contribution to journalLetter

23 Scopus citations
Original languageEnglish (US)
Pages (from-to)e1-e6
JournalHaematologica
Volume102
Issue number1
DOIs
StatePublished - Jan 1 2017
Externally publishedYes

ASJC Scopus subject areas

  • Hematology

Cite this

Saraf, S. L., Shah, B. N., Zhang, X., Han, J., Tayo, B. O., Abbasi, T., Ostrower, A., Guzman, E., Molokie, R. E., Gowhari, M., Hassan, J., Jain, S., Cooper, R. S., Machado, R. F., Lash, J. P., & Gordeuk, V. R. (2017). APOL1,α-thalassemia, and BCL11A variants as a genetic risk profile for progression of chronic kidney disease in sickle cell anemia. Haematologica, 102(1), e1-e6. https://doi.org/10.3324/haematol.2016.154153