Atrial induction of ventricular tachycardia: Reentry versus triggered automaticity

Douglas P. Zipes, Peter R. Foster, Paul J. Troup, Donal H. Pedersen

Research output: Contribution to journalArticle

235 Citations (Scopus)

Abstract

Atrial stimulation induced a sustained ventricular tachycardia in two patients with mitral valve prolapse and in one patient who had mild hypertension without cardiac abnormalities. Exercise-Induced sinus tachycardia also started the ventricular tachycardia in one patient. Evidence is presented to suggest that the mechanism of ventricular tachycardia in one patient was reentrant excitation and in another patient triggered automaticlty. It is likely that the origin of the ventricular tachycardia was confined to a relatively protected small area near the posteroinferior portion of the left ventricle and was not due to macroreentry.

Original languageEnglish
Pages (from-to)1-8
Number of pages8
JournalThe American Journal of Cardiology
Volume44
Issue number1
DOIs
StatePublished - 1979

Fingerprint

Ventricular Tachycardia
Sinus Tachycardia
Mitral Valve Prolapse
Heart Ventricles
Exercise
Hypertension

ASJC Scopus subject areas

  • Cardiology and Cardiovascular Medicine

Cite this

Atrial induction of ventricular tachycardia : Reentry versus triggered automaticity. / Zipes, Douglas P.; Foster, Peter R.; Troup, Paul J.; Pedersen, Donal H.

In: The American Journal of Cardiology, Vol. 44, No. 1, 1979, p. 1-8.

Research output: Contribution to journalArticle

Zipes, Douglas P. ; Foster, Peter R. ; Troup, Paul J. ; Pedersen, Donal H. / Atrial induction of ventricular tachycardia : Reentry versus triggered automaticity. In: The American Journal of Cardiology. 1979 ; Vol. 44, No. 1. pp. 1-8.
@article{b37fb19b483347cbb407ccc1dd45797f,
title = "Atrial induction of ventricular tachycardia: Reentry versus triggered automaticity",
abstract = "Atrial stimulation induced a sustained ventricular tachycardia in two patients with mitral valve prolapse and in one patient who had mild hypertension without cardiac abnormalities. Exercise-Induced sinus tachycardia also started the ventricular tachycardia in one patient. Evidence is presented to suggest that the mechanism of ventricular tachycardia in one patient was reentrant excitation and in another patient triggered automaticlty. It is likely that the origin of the ventricular tachycardia was confined to a relatively protected small area near the posteroinferior portion of the left ventricle and was not due to macroreentry.",
author = "Zipes, {Douglas P.} and Foster, {Peter R.} and Troup, {Paul J.} and Pedersen, {Donal H.}",
year = "1979",
doi = "10.1016/0002-9149(79)90242-X",
language = "English",
volume = "44",
pages = "1--8",
journal = "American Journal of Cardiology",
issn = "0002-9149",
publisher = "Elsevier Inc.",
number = "1",

}

TY - JOUR

T1 - Atrial induction of ventricular tachycardia

T2 - Reentry versus triggered automaticity

AU - Zipes, Douglas P.

AU - Foster, Peter R.

AU - Troup, Paul J.

AU - Pedersen, Donal H.

PY - 1979

Y1 - 1979

N2 - Atrial stimulation induced a sustained ventricular tachycardia in two patients with mitral valve prolapse and in one patient who had mild hypertension without cardiac abnormalities. Exercise-Induced sinus tachycardia also started the ventricular tachycardia in one patient. Evidence is presented to suggest that the mechanism of ventricular tachycardia in one patient was reentrant excitation and in another patient triggered automaticlty. It is likely that the origin of the ventricular tachycardia was confined to a relatively protected small area near the posteroinferior portion of the left ventricle and was not due to macroreentry.

AB - Atrial stimulation induced a sustained ventricular tachycardia in two patients with mitral valve prolapse and in one patient who had mild hypertension without cardiac abnormalities. Exercise-Induced sinus tachycardia also started the ventricular tachycardia in one patient. Evidence is presented to suggest that the mechanism of ventricular tachycardia in one patient was reentrant excitation and in another patient triggered automaticlty. It is likely that the origin of the ventricular tachycardia was confined to a relatively protected small area near the posteroinferior portion of the left ventricle and was not due to macroreentry.

UR - http://www.scopus.com/inward/record.url?scp=0018776615&partnerID=8YFLogxK

UR - http://www.scopus.com/inward/citedby.url?scp=0018776615&partnerID=8YFLogxK

U2 - 10.1016/0002-9149(79)90242-X

DO - 10.1016/0002-9149(79)90242-X

M3 - Article

C2 - 453035

AN - SCOPUS:0018776615

VL - 44

SP - 1

EP - 8

JO - American Journal of Cardiology

JF - American Journal of Cardiology

SN - 0002-9149

IS - 1

ER -