Hepatic venular pressures of rats, dogs, and rabbits

H. Bohlen, R. Maass-Moreno, C. F. Rothe

Research output: Contribution to journalArticle

24 Citations (Scopus)

Abstract

We tested the hypotheses that the hepatic venule pressures (P(hv)), just downstream from the hepatic sinusoids, are closely similar (-1·kg-1) caused P(pv) to increase and the portal venous flow to decrease but did not significantly affect P(hv). The hepatic venous circuit contributed 44 ± 17% (rats) and 31 ± 26% (dogs) of the total liver venous vascular resistance under control conditions. We conclude that the portal and sinusoidal vasculatures are the dominant, but not exclusive, resistance sites of the liver venous vasculature both at rest and during norepinephrine-induced vasoconstriction.

Original languageEnglish (US)
JournalAmerican Journal of Physiology - Gastrointestinal and Liver Physiology
Volume261
Issue number3 24-3
StatePublished - 1991
Externally publishedYes

Fingerprint

Dogs
Rabbits
Pressure
Liver
Venules
Vasoconstriction
Vascular Resistance
Norepinephrine

Keywords

  • Hepatic venules
  • Liver
  • Nonrepinephrine
  • Vascular resistance

ASJC Scopus subject areas

  • Physiology
  • Gastroenterology

Cite this

Hepatic venular pressures of rats, dogs, and rabbits. / Bohlen, H.; Maass-Moreno, R.; Rothe, C. F.

In: American Journal of Physiology - Gastrointestinal and Liver Physiology, Vol. 261, No. 3 24-3, 1991.

Research output: Contribution to journalArticle

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