Role for c-jun N-terminal kinase in treatment-refractory acute myeloid leukemia (AML): Signaling to multidrug-efflux and hyperproliferation

L. D. Cripe, V. M. Gelfanov, E. A. Smith, D. R. Spigel, C. A. Phillips, T. G. Gabig, S. H. Jung, J. Fyffe, A. D. Hartman, P. Kneebone, D. Mercola, G. S. Burgess, H. Scott Boswell

Research output: Contribution to journalArticle

48 Scopus citations
Original languageEnglish (US)
Pages (from-to)799-812
Number of pages14
JournalLeukemia
Volume16
Issue number5
DOIs
StatePublished - Jan 1 2002

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Keywords

  • AML
  • Multidrug resistance
  • Signaling

ASJC Scopus subject areas

  • Hematology
  • Cancer Research

Cite this

Cripe, L. D., Gelfanov, V. M., Smith, E. A., Spigel, D. R., Phillips, C. A., Gabig, T. G., Jung, S. H., Fyffe, J., Hartman, A. D., Kneebone, P., Mercola, D., Burgess, G. S., & Boswell, H. S. (2002). Role for c-jun N-terminal kinase in treatment-refractory acute myeloid leukemia (AML): Signaling to multidrug-efflux and hyperproliferation. Leukemia, 16(5), 799-812. https://doi.org/10.1038/sj.leu.2402457